Androgenetic
Alopecia
The most prevalent form of hair loss worldwide progressive, genetic, and highly treatable when diagnosed early through trichoscopy.
The Most Common
and Most Treatable
Androgenetic alopecia is driven by a combination of genetic predisposition and hormonal sensitivity specifically, the reaction of hair follicles to dihydrotestosterone (DHT). In genetically susceptible individuals, follicles in specific areas of the scalp progressively miniaturise, producing thinner and shorter hair with each cycle until they cease producing visible hair entirely.
The pattern differs by sex: in men, it typically begins with a receding hairline and thinning at the crown. In women, it usually presents as diffuse thinning across the top of the scalp with a noticeably wider parting line, while the frontal hairline is generally preserved.
"It will not resolve on its own but when identified early, the trajectory can be significantly altered. Treatment works best when follicles are miniaturising but still present."
Trichoscopy reveals miniaturisation long before it is visible to the naked eye making early diagnosis possible and treatment more effective.
What to Watch For
Androgenetic alopecia presents differently in men and women recognising the early signs is the first step toward effective intervention.
Thinning at the crown progressing forward the classic male presentation. Often begins as a small patch that gradually expands.
Diffuse thinning across the top of the scalp the typical female pattern. Overall density reduces rather than forming defined bald areas.
Widening of the natural parting line often the first noticeable sign in women. The parting appears broader as density behind it reduces.
Hair becoming progressively finer and shorter with each growth cycle the hallmark of miniaturisation at the follicular level.
Receding temples and hairline typically the earliest visible sign in men, often beginning in the late teens or early twenties.
Noticeable increase in hair shedding during washing or styling particularly when hair that was previously stable begins to fall.
How We Diagnose
Androgenetic Alopecia
High-Resolution Trichoscopy
The definitive clinical tool for diagnosing androgenetic alopecia. Under magnification, we assess follicular density, hair shaft diameter variation, the presence of vellus hairs replacing terminal hairs, and perifollicular discolouration. Trichoscopy identifies miniaturisation before it is visible giving clients the best possible treatment window.
- Follicular density mapping across scalp zones
- Hair shaft calibre measurement and variability assessment
- Staging using Norwood (men) or Ludwig (women) scale
Full Clinical Assessment & Blood Panel
A thorough medical history review covering hormonal history, medications, nutritional status, stress levels, and family history of hair loss. Where hormonal or nutritional factors are suspected, we refer for a targeted blood panel ensuring we address the full picture rather than treating symptoms in isolation.
- DHT, thyroid panel, ferritin, vitamin D when indicated
- Hormonal profile for female pattern hair loss
- Baseline photography for objective progress tracking
How We Treat
Androgenetic Alopecia
The most effective protocols combine multiple biological approaches each targeting a different aspect of the condition.
First Line
Concentrated growth factors from your own blood stimulate miniaturising follicles and improve scalp microcirculation. Most effective at early to moderate stages.
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Advanced Cases
10 billion exosomes deliver targeted regenerative signals directly to follicular cells. 70–90% clinical success rate. Ideal for moderate to advanced cases or PRP non-responders.
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Adjunct
Bioactive peptides and growth factors delivered into the perifollicular layer nourishing miniaturising follicles and complementing PRP or exosome protocols.
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Maintenance
Photobiomodulation stimulates follicular metabolism and may reduce DHT sensitivity. Used as standalone therapy or to extend the results of injectable treatments.
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Advanced Loss
For advanced androgenetic alopecia SMP creates the appearance of a defined, natural shaved-head look or adds visual density to thinning areas. Immediate, permanent result.
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Maintenance
Photobiomodulation stimulates follicular metabolism and may reduce DHT sensitivity. Used as standalone therapy or to extend the results of injectable treatments.
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What You Can
Realistically Expect
With the right protocol, introduced at the right time, androgenetic alopecia can be effectively managed slowing or halting progression, strengthening existing hair, and in many cases, recovering meaningful density.
- Measurable reduction in shedding within 2–3 months of treatment
- Visible density improvement in areas of early miniaturisation
- Halting or significantly slowing further progression
- Results are most significant in early to moderate stages acting before follicles are permanently lost is the single most important factor
Diagnosed early,
treated effectively.
The most important thing you can do if you suspect androgenetic alopecia is to get a clinical assessment not to wait and see.
